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On the other hand, it further exacerbates ferroptosis through changes to the metabolite profilesspecifically, reducing the production of anti-ferroptosis metabolites such as 5-HT and 3-HAA, and increasing the abundance of pro-ferroptosis metabolites such as LPS and TMAO

These cardiovascular adverse effects include first-dose bradycardia, atrioventricular conduction block and hypotension, and they must be cautiously considered when utilizing this drug for CIPN prevention and treatment [4]

This metabolic shift enables MDSCs to sustainably generate energy in hypoxic environments, while concurrently mitigating ROS production and subsequent apoptosis, thereby safeguarding their survival and functionality (110, 112).Chiu et al

doi:10.1126/science.289.5484.1567