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77 Thus, the KD could regulate dysregulated brain metabolism, mitochondrial homeostasis, and inflammation in AD

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Grice EA, Kong HH, Renaud G, et al

Figure 2 Both insoluble fibrils and soluble A oligomers exert potent neurotoxicity by forming plaques and triggering pathological cascades, including synaptic failure, excitotoxicity, changes in membrane permeability, and chronic inflammation (77, 78)