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SREBP1c ablation also promotes a compensatory upregulation of SREBP2, leading to increased hepatic cholesterol synthesis and cholesterol accumulation, linking DNL to cholesterol metabolism [46]

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Their findings suggested that NAC disrupts vWF multimers via disulfide bond reduction, reducing platelet aggregation and microthrombi formation, which are central to sepsis-induced organ failure (Liu et al., 2025)