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This patent describes covalent immobilization of the N-terminal intein fragment and provides high ligand densities with the potential to reuse the resin after multiple harsh cleaning procedures

This study also determined the role of GSH in PfK13-mediated resistance and revealed 1) that modulation of PfK13 levels or localisation modifies GSH levels, and (2) that elevated GSH decreases artemisinin potency using in vitro ring-stage survival assays

Researchers requiring separate experimental assessment of each peptide may alternatively utilize the individual preparations.