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Although GSH supplementation partially increased viability in both groups, koSLC25A39 cells exhibited persistently reduced viability compared to controls, indicating that exogenous GSH could not fully rescue the viability deficit caused by loss of the mitochondrial inner membrane GSH transporter SLC25A39 (Fig

Our study indicated that ten cycles of 10-s reperfusion and 10-s ischemia effectively attenuated testis damage after 24 h

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Additionally, extracellular calcium influx and subsequent Ca 2+ uptake by mitochondria were both required for mitochondrial ROS production ( DRAK2 as a an Intermediary in Calcium and Metabolic Signaling in T Cells One molecule expressed in T cells that may potentially link calcium and ROS signaling to cellular metabolic regulation is DRAK2/STK17B