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Rodrguez Cetina Biefer H, et al

This was exemplified here by the fact that increased GSH production in the presence of 5-FOA (counter-selective for URA3 expression) appeared to be a non-specific response to 5-FOA, evident also in the control strain expressing URA3 behind pURA3 , rather than selection based on phenotypic heterogeneity as intended in this proof-of-principle study

Uncovering the Role of N-Acetyl-Aspartyl-Glutamate as a Glutamate Reservoir in Cancer

These are the DNA nitrogen bases, the DNA nitrogen base pairs, certain free radicals which are responsible for oxidative stress and GSH which is apparently important as it is involved in the genotoxic activity of copper compounds [52-58]

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