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Strategic manipulation of intrinsic pathways, exemplified by Phosphatase and Tensin homolog (PTEN) inhibition, can suppress growth constraints and re-activate robust axonal extension [267, 268]

Thus, one consequence of oxidative damage to mitochondrial membranes and proteins is the uncoupling of electron transport and ATP production resulting in an increase in mitochondrial release of ROS (mostly as hydrogen peroxide, although some superoxide may also be released) [15]

Do schizophrenia patients make more perseverative than non-perseverative errors on the Wisconsin Card Sorting Test