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For instance, one study has demonstrated that endogenous DNMT3A and DNMT3B are associated with HDAC activity, where DNMT3A facilitates transcriptional repression via specific molecular interactions between its ATRX-like PHD domain and both HDAC1 and the co-repressor RP58 (302)

Toxic doses in research have been measured at 300-3,750 times higher than therapeutic ranges, providing substantial safety margins

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Generally, p53 is considered as a stress responsive protein that activates different downstream pathways depending on the magnitude, duration, and type of upstream stressors