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Hence, after vB12 treatment, increased GABA, together with the restoration of normal (low) levels of glutamate, might counteract the potentially neurotoxic excitatory effects of increased glutamate in the Tbx1 +/ brain

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However, it is noteworthy that whereas there are trends showing significant association between MQ and N86Y over the study period, none can be established between LU and N86Y (Figure 2D)