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Furthermore, Nalm6 cells lacking FANCB were significantly sensitive to GSH inhibition even in the absence of FA, which might be a consequence of an increase in endogenous free FA, overall suggesting that GSH supply might be fundamental for Fanconi Anaemia patients (Fig

23 Indeed, directly targeting Ras using, among other agents, the farnesyltransferase inhibitor tipifarnib has been extremely challenging in general 24 and was also demonstrated to be ineffective in the treatment of pNF 25 (NCT00021541)

Patients with decreased GSH and increased oxidative stress also showed increased neutrophil influx and IL-8 levels (101)

The -tubulin loading control shown is identical to that in Figure 2O, as the same protein lysates were analyzed