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10.1021/acsnano.9b00300 60 WangB.DaiY.KongY.DuW.NiH.ZhaoH.et al (2020)

A proposed mechanistic explanation is that FATP5, by preferentially channeling long-chain fatty acids toward mitochondrial -oxidation rather than membrane biosynthesis or signaling lipid pools, may paradoxically limit the availability of lipid substrates for oncogenic signaling

Plants [edit source] In higher plants, superoxide dismutase enzymes (SODs) act as antioxidants and protect cellular components from being oxidized by reactive oxygen species (ROS)