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Description

Neutralizing antibodies to IL-18 ameliorate experimental autoimmune neuritis by counter-regulation of autoreactive Th1 responses to peripheral myelin antigen

2.2.1 An age-related sensitization of NLRP3 expression amplifies low-grade inflammation Age-related declines in homeostatic mechanisms and dietary accumulations contribute to the increased presence of endogenous components, serving as priming signals for NLRP3