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Given that global or neural-specific GLUT3 knockout models result in embryonic lethality or neurobehavioral abnormalities 82,83,84 , we used a GLUT3 KD approach in N41 cells to investigate its functional role

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It reduces discomfort without increasing the risk of bleeding, unlike some pain relievers such as ibuprofen or aspirin

It provides a logical explanation for the development of dual targeting drugs such as MM-131 a bispecific anti-MET/EpCAM mAb [108] and also further drug discoveries targeting concurrently all three c-Met, EpCAm and Wnt pathway simultaneously to overcome both innate and acquired resistance to tyrosin kinase inhibitors (TKIs)