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Interestingly, the anti-VEGFR2 antibody (DC101) therapy increases the expression of CX3CL1 that induce Ly6C lo monocytes infiltration, in turn these cells produce CXCL5 that induces immunosuppressive Ly6G+ neutrophils recruitment as a resistance mechanism [49]

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Some of these include the use of organs from older donors and grafts such as small-for-size or steatotic livers