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In addition to iron uptake, after crossing the outer membrane, zinc uptake is facilitated by a periplasmic binding protein and a homologous membrane permease system ZnuABC (also called MntABC in the literature), and mutants unable to produce any of these proteins are defective in intracellular survival (Lim et al., 2008)

This uptake influences ATP synthesis, as calcium serves as a cofactor for mitochondrial dehydrogenases in the TCA cycle

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