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After TBI, there is an activation of neuroinflammation response pathway mediated by microglia cells that were not initially damaged, and the activated microglial cells may promote the release of proinflammatory factors such as interleukin 1, tumor necrosis factor, nitric oxide, etc., (Kumar and Loane, 2012), inducing a progressive cycle of chronic inflammation (Sulhan et al., 2020)

It reduces mucus and inflammation from the airway during lung detoxification

A positive correlation, as found between GSH production and arginine, denotes that a higher flux rate through the exchange reaction of the metaboliteless uptake or more secretionenables the model to produce GSH more rapidly

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