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More specifically, these rats showed dose-dependent increases in anti-apoptotic mediators (like BCL2) and decreases in pro-apoptotic mediators (like BAX and caspase-3) as well as increased concentrations of antioxidants like superoxide dismutase and catalase [48]

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Age-related cognitive impairment is associated with long-term neuroinflammation and oxidative stress in a mouse model of episodic systemic inflammation