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Numerous studies have focused on the cell pathology of the cerebella involving Purkinje cells for ASD [144]

Therefore, the mechanism by which AS-IV inhibits MF may be attributed to regulatory effects on oxidative stress and the release of aging markers (62-64)

Its contribution to embryonic development, maintenance of homeostasis, as well as involvement in other basic physiological processes and pathological situations is well documented [73]

187 Therefore, EV miRNAs may play a dual role in the aging process: on the one hand, they stimulate cells and tissues to become more dynamic and delay aging, and on the other hand, they drive metabolic insufficiency to aggravate aging