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The expression of NMR-1 using the heterologous glr-1 promoter helped us to exclude the possible involvement of the germline in this GSSG-mediated learned aversion because the glr-1 gene is expressed in most NMR-1-expressing interneurons, such AVA, AVD, and AVE neurons 61 , though not in the germline

D., Stinnett, G

IGF-1 levels increase significantly but remain within safety thresholds, and no glycemic worsening occurs in type 2 diabetes patients at therapeutic doses