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AD-GTPCHD is also called autosomal dominant Segawa syndrome (DYT5a), whereas autosomal recessive Segawa syndrome is usually caused by autosomal recessive mutations of the TH gene (DYT5b)

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These observations have made BPC-157 a useful research tool for examining peptide-mediated signal transduction, cytoskeletal organization, extracellular matrix dynamics, and the coordination of complex cellular signaling networks