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doi: 10.1016/j.bbrc.2020.05.015 49

At the molecular level, BPC-157 activates VEGFR2 signaling to drive new vessel formation at wound sites, mobilizes FAK-paxillin complexes that govern cell adhesion and directional migration, and upregulates growth hormone receptor expression in fibroblasts dramatically amplifying the healing cascade

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[10] The mechanism of the quantitative assay is mainly based on light absorption