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Moreover, GH signaling to elicit IGF1 gene transcription is blocked in the absence of appropriate insulin signaling, a phenomenon also known as un-coupling, and resulting in growth arrest (60, 61, 63, 64) (Figure 3)

In the same study hesperetin (10 M) enhanced mitochondrial function by promoting ATP synthesis and mitochondrial share capacity in in vitro model of human differentiated myotubes, suggesting this agent as possible alternative for sarcopenia and retardation of mitochondrial dysfunction

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