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10.1152/ajplung.00073.2014 134 ShahidiF.ZhongY

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Further molecular refinement revealed the enrichment of missense mutations and deletions in histone-lysine N-methyltransferase 2A ( KMT2A ) or MLL and histone deacetylase ( HDAC ) family genes within Cluster M2, underscoring the role of chromatin remodeling in GBM pathogenesis