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On the other hand, STING1-mediated autophagy and MFN1 (mitofusin1)-MFN2-dependent mitochondrial fusion can increase ferroptosis sensitivity to various stimuli, including GPX4 inhibitors [186]

F.GomesG

Ismaiel, M

In the transition from the occluded to the inward conformational state, TM1a is dilated away from the transporter core, thus opening up the intracellular pathway for ion release and then substrate release