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A basic study suggested that chidamide inactivates Hh signaling by increasing the level of miR-338-5p, increasing oxidative stress and promoting glioma cell apoptosis and necrosis (Zhou et al., 2020)

Moreover, to achieve personalized treatment, we need to delve deeper into the interactions between leukemia-specific autophagy and metabolism, and develop biomarkers based on autophagy for precise patient stratification

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