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Follicles exposed to AHK-Cu at 10 to the negative ninth molar showed significantly greater elongation compared to untreated controls

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Mito-Met 10 ), with enhanced mitochondrial targeting capacity, have been demonstrated to enhance metformin-mediated inhibition of mitochondrial respiration and radiosensitisation in prostate cancer ( in vitro and ex vivo models of oesophageal adenocarcinoma ( We also demonstrate metformin-mediated alterations to cell cycle distribution and progression following radiation in HCT116 and SW837 cells, with metformin demonstrated to reduce the proportion of G2/M phase cells and prevent radiation-induced G2/M blockade in these cells