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CPZ intoxication mimics several degenerative events in the brain, inducing demyelination, loss of OLs, axonal and synaptic dysfunction, glial cell activation, and oxidative stress, recapitulating cellular and molecular degenerative features of demyelinating diseases 31,32,33,34

Activation of human microglia by fibrillar prion protein-related peptides is enhanced by amyloid-associated factors SAP and C1q

Despite the availability of alternative diagnostic modalities for nerve injury assessment, most remain validated against NCS as the clinical reference standard

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